Atherosclerosis is considered to be a T-cell-driven disease since the interaction between resident cells, immune cells and their products has been recognized to promote the development of atheromatous plaques in the arterial walls [1]. The process of atherosclerosis evolves from a prelesional phase, which is potentially reversible, to complicated lesions characterized by the formation of a fibrous cap and vulnerable plaques, followed by possible rupture and atherothrombosis.

Gelsomino, F., Fiorentino, M., Zompatori, M., Poerio, A., Melotti, B., Sperandi, F., et al. (2018). Programmed death-1 inhibition and atherosclerosis: Can nivolumab vanish complicated atheromatous plaques?. ANNALS OF ONCOLOGY, 29(1), 284-285 [10.1093/annonc/mdx718].

Programmed death-1 inhibition and atherosclerosis: Can nivolumab vanish complicated atheromatous plaques?

Gelsomino, F.;Fiorentino, M.;Zompatori, M.;Poerio, A.;Melotti, B.;Gargiulo, M.;Borghi, C.;Ardizzoni, A.
2018

Abstract

Atherosclerosis is considered to be a T-cell-driven disease since the interaction between resident cells, immune cells and their products has been recognized to promote the development of atheromatous plaques in the arterial walls [1]. The process of atherosclerosis evolves from a prelesional phase, which is potentially reversible, to complicated lesions characterized by the formation of a fibrous cap and vulnerable plaques, followed by possible rupture and atherothrombosis.
2018
Gelsomino, F., Fiorentino, M., Zompatori, M., Poerio, A., Melotti, B., Sperandi, F., et al. (2018). Programmed death-1 inhibition and atherosclerosis: Can nivolumab vanish complicated atheromatous plaques?. ANNALS OF ONCOLOGY, 29(1), 284-285 [10.1093/annonc/mdx718].
Gelsomino, F.; Fiorentino, M.*; Zompatori, M.; Poerio, A.; Melotti, B.; Sperandi, F.; Gargiulo, M.; Borghi, C.; Ardizzoni, A.
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Utilizza questo identificativo per citare o creare un link a questo documento: https://hdl.handle.net/11585/621334
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