The phytoplasma aetiology of epidemic stunting of cladodes and stunted growth observed in a cactus pear plantation in Carlentini (Sicily, Italy) was investigated with graft inoculation trials and PCR/restriction fragment length polymorphism analyses of 16S ribosomal RNA (rRNA) gene and S3 ribosomal protein gene. After sequencing of the 1525-bp from 16S rRNA gene of both naturally infected and graft-inoculated symptomatic cactus pear samples, phytoplasma TS belonging to ribosomal subgroup 16SrII-C were identified as aetiological agents of this worldwide spread disease.
Granata G., S. Paltrinieri, S. Botti, A. Bertaccini. (2006). Aetiology of Opuntia ficus-indica malformations and stunting disease. ANNALS OF APPLIED BIOLOGY, 149, 317-325 [10.1111/j.1744-7348.2006.00097.x].
Aetiology of Opuntia ficus-indica malformations and stunting disease.
PALTRINIERI, SAMANTA;BOTTI, SIMONA;BERTACCINI, ASSUNTA
2006
Abstract
The phytoplasma aetiology of epidemic stunting of cladodes and stunted growth observed in a cactus pear plantation in Carlentini (Sicily, Italy) was investigated with graft inoculation trials and PCR/restriction fragment length polymorphism analyses of 16S ribosomal RNA (rRNA) gene and S3 ribosomal protein gene. After sequencing of the 1525-bp from 16S rRNA gene of both naturally infected and graft-inoculated symptomatic cactus pear samples, phytoplasma TS belonging to ribosomal subgroup 16SrII-C were identified as aetiological agents of this worldwide spread disease.I documenti in IRIS sono protetti da copyright e tutti i diritti sono riservati, salvo diversa indicazione.
